| For medical practitioners and the general public - High Blood Pressure Journal Article Catalog. |
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| Diastolic blood pressure responses following direct and displaced aggression after anger arousal in high- and low-guilt subjects Gambaro, S. and A. I. Rabin (1969), J Pers Soc Psychol 12(1): 87-94. |
| Dietary sodium and blood pressure in young people with and without familial predisposition to high blood pressure Watt, G. C., C. J. Foy, et al. (1986), J Clin Hypertens 2(2): 141-7. |
| Different effects of aspirin on blood pressure of spontaneously hypertensive rats (SHR) with high and spontaneously low levels of blood pressure Schirner, M. and C. Taube (1993), Br J Pharmacol 109(4): 900-1. Abstract: Spontaneously hypertensive rats (SHR) of the Okamoto strain with blood pressure above 161 mmHg and SHR with blood pressure levels of less than 160 mmHg were treated with oral doses of aspirin (100 mg kg-1) for three days. Whereas the blood pressure of SHR with blood pressure above 161 mmHg was decreased by aspirin, the blood pressure of SHR below 160 mmHg was increased by aspirin. The extent and direction of blood pressure change by aspirin was strongly correlated with the blood pressure of SHR before treatment (r = -0.88). The effect of aspirin supports an important role for endogenous prostanoids in the regulation of blood pressure of SHR. |
| Different mechanisms maintaining high blood pressure in chronic one-kidney, one-clip, and two-kidney, one-clip hypertensive rats Hashimoto, H., K. Hiwada, et al. (1983), Clin Exp Hypertens A 5(3): 429-45. Abstract: To evaluate the difference of the blood pressure regulating mechanisms of chronic (12-14 weeks) one-kidney, one-clip (1K-1C) and chronic two-kidney, one-clip (2K-1C) hypertensive rats, we administered captopril, captopril plus indomethacin, and indomethacin to the rats. Pretreatment values of plasma renin concentration, plasma aldosterone concentration and urinary kallikrein excretion were significantly higher in 2K-1C than in 1K-1C hypertensive rats. Captopril-induced blood pressure reduction was greater in 2K-1C than in 1K-1C hypertensive rats. When captopril was administered to the rats treated with indomethacin, captopril-induced blood pressure reduction was attenuated only in 2K-1C hypertensive rats. Indomethacin produced renal impairment and further raised the blood pressure in 1K-1C hypertensive rats, but did not in 2K-1C hypertensive rats. These results suggest that the renin-angiotensin system functions to maintain high blood pressure more predominantly in chronic 2K-1C than in 1K-1C hypertensive rats. The renal kallikrein-kinin system is suppressed in chronic 1K-1C hypertensive rats but not in 2K-1C hypertensive rats. The renal prostaglandin system is more important for regulating the renal circulation in chronic 1K-1C than in 2K-1C hypertensive rats. |
| Different plasma ionized calcium correlations with blood pressure in high and low renin normotensive adults in Utah Hunt, S. C., R. R. Williams, et al. (1991), Am J Hypertens 4(1 Pt 1): 1-8. Abstract: Plasma ionized calcium levels have been shown to be lower than normotensive control levels in hypertensive patients with low plasma renin activity and higher than control levels in hypertensive patients with high renin activity; they did not differ between high and low plasma renin activity groups of normotensive controls. To see if ionized calcium may have different relationships with blood pressure across renin categories in normotensive individuals, plasma ionized calcium was measured on 875 healthy individuals, ages 3 to 83, who had never been diagnosed as having hypertension. Blood pressures were measured in the sitting, standing, and supine positions, along with pressures measured during two stress maneuvers: isometric handgrip and a 50 degrees tilt from a supine position. There was no linear correlation of blood pressure with plasma ionized calcium in the entire sample of youths or adults. However, after dividing the adults into tertiles based on plasma renin activity, there were significant inverse correlations between ionized calcium and systolic and diastolic blood pressure in the low renin group (r = -0.16 to -0.25, P less than or equal to.05), while the systolic blood pressure correlations were significantly positive in the high renin group (r = 0.14 to 0.22, P less than or equal to.05). Adults with normal renin levels did not have any significant correlations of plasma ionized calcium with blood pressure. These confounding effects of renin were greater for systolic than for diastolic blood pressure. These correlations within renin tertiles occurred even though there were no differences in mean blood pressure, plasma ionized calcium, total plasma protein and plasma sodium across renin categories.(ABSTRACT TRUNCATED AT 250 WORDS) |
| Differential diagnosis of high blood pressure Fishberg, A. M. (1954), Med Clin North Am New York No.: 753-64. |
| Differential impact of systolic and diastolic blood pressure level on JNC-VI staging. Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure Lloyd-Jones, D. M., J. C. Evans, et al. (1999), Hypertension 34(3): 381-5. Abstract: The sixth Report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure classifies blood pressure into stages on the basis of both systolic (SBP) and diastolic (DBP) blood pressure levels. When a disparity exists between SBP and DBP stages, patients are classified into the higher stage ("up-staged"). We evaluated the effect of disparate levels of SBP and DBP on blood pressure staging and eligibility for therapy. We examined 4962 Framingham Heart Study subjects between 1990 and 1995 and determined blood pressure stages on the basis of SBP alone, DBP alone, or both. After the exclusion of subjects on antihypertensive therapy (n=1306), 3656 subjects (mean age 58+/-13 years; 55% women) were eligible. In this sample, 64.6% of subjects had congruent stages of SBP and DBP, 31.6% were up-staged on the basis of SBP, and 3.8% on the basis of DBP; thus, SBP alone correctly classified JNC-VI stage in approximately 96% (64.6%+31.6%) of the subjects. Among subjects >60 years of age, SBP alone correctly classified 99% of subjects; in those |
| Differential therapeutic aspects of high blood pressure therapy with beta blockers Buhler, F. R. and P. Bolli (1983), Schweiz Rundsch Med Prax 72(20): 684-90. |
| Differential therapeutic principles in the drug therapy of high blood pressure Rahn, K. H. (1974), Internist (Berl) 15(3): 157-64. |
| Diminished baroreflex sensitivity in high blood pressure Bristow, J. D., A. J. Honour, et al. (1969), Circulation 39(1): 48-54. |
| Diminished baroreflex sensitivity in high blood pressure and ageing man Bristow, J. D., B. Gribbin, et al. (1969), J Physiol 202(1): 45P-46P. |
| Diminished pulse pressure under mental stress characterizes normotensive adolescents with parental high blood pressure Ewart, C. K., W. L. Harris, et al. (1986), Psychosom Med 48(7): 489-501. Abstract: An exaggerated blood pressure response to mental stress is believed to characterize young adults with genetic risk of essential hypertension, suggesting that stress-induced changes might provide a useful index of pathogenetic processes. We explored this by studying pressor responsivity to competitive tasks in adolescents drawn from a large urban population. Individuals with systolic or diastolic pressures persistently between the 85th and 95th percentiles were evaluated on basal blood pressure, parental history of hypertension, and pressor and heart rate response to a challenging video game. Basal pressure was measured again at 6, 10, and 14 months. A persistently diminished pulse pressure was the cardiovascular characteristic that most reliably typified normotensive subjects with familial hypertension. Response to the video game was the best indicator of risk status. Contrary to expectations derived from research with convenience samples, epidemiologic investigation points to an increased peripheral resistance and lower cardiac output as the cardiovascular pattern more prominently associated with genetic risk in the normotensive adolescent. |
| Directory of high blood pressure resources available to physicians Lewis, C., E. C. Amador, et al. (1984), Md State Med J 33(3): 216-23. |
| Discourses of worry, stress, and high blood pressure in rural south Louisiana Boutain, D. M. (2001), J Nurs Scholarsh 33(3): 225-30. Abstract: PURPOSE: To explore how a sample of rural Louisiana residents constructed accounts about worry and stress in relationship to their high blood pressure. DESIGN: Qualitative study combining critical social theories, African American studies, and critical discourse concepts. Study participants consisted of a convenience sample (N = 30) of African American women (n = 15) and men (n = 15) with high blood pressure. METHODS: Over a 4-month period in 1999 a community-based population sample was interviewed twice. Field experiences in the community and the assistance of community consultants were critical to data analysis. Based on 60 interviews, 191 passages about worry and 58 passages about stress were analyzed using discourse analysis. FINDINGS: Participants not only distinguished between worry and stress in their everyday lives, but they also highlighted how those concepts were interrelated. Participants' concerns about themselves as well as their children, kin, and community were emphasized in passages about worry. Stress was primarily associated with doing multiple tasks and confronting multiple prejudices in the workplace and surrounding community. CONCLUSIONS: Participants perceived worry and stress as important health-related concepts that affected their high blood pressure. Nursing strategies designed to address these concerns may better facilitate holistic health. |
| Discrimination learning in mice genetically selected for high and low blood pressure: initial findings and methodological implications Elias, M. F. and G. Schlager (1974), Physiol Behav 13(2): 261-7. |
| Diseases of the urinary system. High blood pressure and renal disease MacGregor, G. A. (1977), Br Med J 2(6087): 624-6. |
| Dissociation between derepressed K+,Cl- cotransport system and high blood pressure in the F2 hybrid generation (SHR x WKY) Rota, R., C. Nazaret, et al. (1991), J Hypertens Suppl 9(6): S298-9. |
| Distribution of blood pressure and hypertension in healthy subjects residing at high altitude in the Himalayas Puri, D. S., L. S. Pal, et al. (1986), J Assoc Physicians India 34(7): 477-9. |
| Distribution of diffusible and non-diffusible calcium in the blood plasma of Anodonta determined by high pressure ultrafiltration. Schoffeniels, E. (1951), Arch Int Physiol 59(1): 49-52. |
| Distribution of pyrrolidinomethyl-tetracycline (rolitetracycline) and tetracycline in blood and various organs of mice measured by high pressure liquid chromatography Bocker, R. and C. J. Estler (1979), Arzneimittelforschung 29(11): 1693-5. Abstract: By means of a newly developed high-pressure liquid chromatographic method the organ distribution of tetracycline (TC) and pyrrolidinomethyl-tetracycline (PMT) has been studied. When mice were treated with 50 mg/kg i.v. TC or PMT these antibiotics could be detected in all organs investigated (liver, kidney, heart, lung, muscle, spleen). Especially high concentrations were found in liver and kidneys, where TC and PMT could be detected up to 6 h. In animals treated with PMT part of the PMT applied decomposed slowly yielding TC, which was found together with PMT in blood and all organs. |
| Divergent responses of common carotid artery intimal-medial thickness to high blood pressure and hypercholesterolaemia in relation to age Ludwig, M. M., U. Jorger, et al. (1993), J Hypertens Suppl 11(5): S130-1. |
| Do angiotensin II receptor antagonists substitute angiotensin converting enzyme inhibitors in the treatment of high blood pressure? Gonzalez-Juanatey, J. R. (2000), Rev Esp Cardiol 53(1): 4-12. Abstract: Angiotensin converting enzyme inhibitors (ACEI) and angiotensin II receptor antagonists (AIIA) are both pharmacological groups that inhibit the actions of angiotensin II. ACEI prevent the formation of angiotensin II from angiotensin I, whereas A II A inhibit the final crucial step of angiotensin II binding with the AT1 receptor site. A similar antihypertensive efficacy has been described for both groups but A II A drugs have a better safety profile above all due to the absence of dry cough. Despite the fact that evidence with ACEI is more conclusive, A II A seems to achieve the same protective effects on the target organ damage in hypertensive patients. At present, ACEI are the drugs of choice in the treatment of patients with cardiac dysfunction and failure. The information of ongoing trials with A II A will be of great value in deciding the optimal treatment for hypertensive patients with different cardiovascular diseases. |
| Do subjects with stiff arteries have high blood pressure? Cox, J. P., R. England, et al. (1989), J Hypertens Suppl 7(6): S82-3. Abstract: It has been argued that age-related increases in arterial stiffness could lead to spuriously high indirect blood pressure measurements, with consequent overdiagnosis of hypertension in older patients. To study the relationship between arterial stiffness and blood pressure, we identified patients with 'arterial stiffness', using Osler's manoeuvre, and compared their blood pressure levels with patients of a similar age. A total of 250 hospital inpatients were assessed independently by two doctors. In the 198 patients (79%) where both observers agreed on Osler's manoeuvre status, positive Osler's manoeuvre was uncommon under the age of 50 years but became more common thereafter, rising to 58% of patients aged over 75 years. However, blood pressure levels were similar in each age group, irrespective of Osler's manoeuvre status. We conclude that increased arterial stiffness as measured by Osler's manoeuvre is not necessarily associated with raised blood pressure levels in the elderly. |
| Does a high P/S ratio diet lower blood pressure? Margetts, B. M. (1990), Klin Wochenschr 68 Suppl 20: 11-5. Abstract: The answer to the question the title of this paper puts is: No a P/S ratio diet alone does not lower blood pressure! Although a number of cross-sectional and other studies do suggest that there is a relationship between P/S ratio and blood pressure it is not possible in these studies to eliminate the potentially confounding effects of other aspects of diet and lifestyle which are also either different or affected by the experiment. Most of the evidence from properly controlled randomised experimental studies do not provide support for an effect of an elevation of the dietary P/S ratio alone on blood pressure. These experiments have been conducted at various levels of total fat intake, across a range of blood pressures (but not very high blood pressures) and for both a short or longer duration. The levels of polyunsaturated fats have also varied from modest increases to quite substantial increases. It is therefore unlikely that aspects of sample selection, range of dietary change or length of observation can account for the failure of these studies to show an effect on blood pressure. From other observational and experimental studies it is apparent that dietary change can affect blood pressure. It may be that combinations of dietary factors need to occur in synchrony to affect blood pressure. It is therefore possible that a high P/S ratio diet in combination with for example potassium, calcium or magnesium or other dietary factors may influence blood pressure.(ABSTRACT TRUNCATED AT 250 WORDS) |
| Does extreme dipping of nocturnal blood pressure in elderly hypertensive patients confer high risk of developing ischemic target organ damage from antihypertensive therapy? Kario, K. and T. G. Pickering (2000), Arch Intern Med 160(9): 1378. |
| Does oral calcium supplementation lower high blood pressure? A double blind study Cappuccio, F. P., N. D. Markandu, et al. (1987), J Hypertens 5(1): 67-71. Abstract: Eighteen unselected patients with untreated mild to moderate essential hypertension, whose average supine blood pressure after 2 months' observation on no treatment was 154/103 mmHg, were entered into a double-blind randomized crossover study of 1 month's treatment with calcium lactate gluconate (40 mmol of elemental calcium/day) and treatment with placebo for a further month. Despite a significant increase in total plasma calcium (P less than 0.01) and in 24-h urinary excretion of calcium (P less than 0.025) while taking calcium lactate gluconate, there was no fall in blood pressure with calcium supplementation compared to treatment with placebo. |
| Doing something about high blood pressure Robertson, J. I. (1978), Indian J Med Sci 32(3-4): 35-7. |
| Dopamine infused continuously at high concentration with a low flow rate affects arterial blood pressure fluctuation waves Shibata, H., M. Aibiki, et al. (1993), Crit Care Med 21(5): 801-4. |
| Dormont High School (Pittsburgh, Pennsylvania) blood pressure study Kuller, L. H., M. Crook, et al. (1980), Hypertension 2(4 Pt 2): 109-16. Abstract: A cohort of high school students examined in a school health study between 1957-63 were followed to 1977, when as adults still living in the area, 373 (71%) were reexamined. Mean age at 17-year follow-up was 34 years. The mean systolic blood pressure (SBP) at follow-up for men was 125 mm Hg, women 111 mm Hg. The boys' SBP had increased 4.0 Hg while the girls' declined 4.0 mm Hg in 17 years. The boys had gained an average of 37.2 lbs, and 1 inch, the girls 16.7 lbs, and 0.5 inch. Tracking was studied in several ways. The correlation coefficient of the SBP taken 17 years apart ws 0.44 for boys and 0.39 for girls. Current SBP was 115 mm Hg for boys with the lowest tenth of high school SBPs and 131 mm Hg for the boys in the highest tenth. Thirty-nine had hypertension, DBP greater than or equal to 90, or were on antihypertensive medication. They had had substantially higher SBP and weight in high school, and had gained more weight from high school to adult life than controls. After adjusting for high school SBP, weight gain for boys was the major determinant of subsequent high blood pressure (BP). |
| Down with high blood pressure Chaudhuri, B. R. (1978), J Indian Med Assoc 70(7): 166-7. |
| Down with high blood pressure Isacsson, S. O., N. Metsnaa, et al. (1978), Lakartidningen 75(14): 1381. |
| Down with high blood pressure Lovell, R. R. (1978), Med J Aust 1(7): 365-6. |
| Down with high blood pressure! Mahler, H. (1978), CICIAMS Nouv(1): 3-4, 10-1. |
| Down with high blood pressure, World Health Day, 7 April 1978: hypertension in Africa Akinkugbe, O. O. (1978), Indian J Med Sci 32(5-6): 61-4. |
| Down with high blood pressure: World Health Day, 7 April 1978. Cuba fights hypertension Castro, I. M. (1978), Indian J Med Sci 32(7-8): 83-5. |
| Dried blood spot Vitamin A determination by high pressure liquid chromatography with electrochemical detection Houze, P., S. Beltz, et al. (2004), Ann Biol Clin (Paris) 62(5): 539-46. Abstract: In tropical countries. vitamin A deficiency is one of the most important dietary deficiencies. Its monitoring usually involves analysis of retinol after venipuncture with some difficulties (disease transmission, religious belief). Sample collection on Dried Blood Spot (DBS) is less invasive and safer. Sample storage is easier. We developed a liquid chromatography method with electrochemical detection to measure DBS retinol. Retinol acetate was used as an internal standard. The method is linear up to 2.5 microM with a detection limit of 0.04 microM. Precision is below 10% and DBS retinol recovery overage is 90%. DBS retinol concentration decreased during 7 days after sampling, it is necessary to wait this delay before to determine vitamin A concentrations. In Congolese children DBS retinol measurement showed a severe vitamin A deficiency in 8% of them. This percentage is closely correlated with clinical parameters. |
| Drug information. The use of captopril in high blood pressure Volmer, M. (1983), Tijdschr Ziekenverpl 36(18): 573-5. |
| Drug Therapy Of High Blood Pressure In The Past, Present And Future. Mathisen, H. S. (1963), Tidsskr Nor Laegeforen 83: 1679-80. |
| Drug therapy today: drugs for managing high blood pressure Rodman, M. J. (1969), Rn 32(5): 73-80. |
| Drug treatment of high blood pressure Hutchins, L. N. (1981), Nurs Clin North Am 16(2): 365-76. Abstract: The effective drug treatment of hypertension has become a reality over the past 25 years. People no longer need die from complications of untreated hypertension. Pharmacologic treatment is available with a variety of antihypertensive drugs: diuretics, central nervous system agents, alpha-adrenergic blockers, beta-adrenergic blockers, vasodilator, and new drugs that are still under investigation. The desired outcome of a lowered blood pressure can usually be achieved by the stepped-care approach coupled with nonpharmacologic measures. Individualization of therapy, however, is essential. |